By Bruce E. Murdoch
The long-held trust that got aphasia in childrens is basically of the non-fluent kind has been challenged in recent times. This e-book discusses language difficulties bobbing up from cerebro-vascular injuries taking place in adolescence, and from different
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Additional resources for Acquired Neurological Speech Language Disorders In Childhood (Brain Damage, Behaviour and Cognition)
A suggestion of the presence of pragmatic problems, however, can be gleaned from earlier writings which suggested that soon after the onset of aphasia or in connection with the mute phase, children with acquired aphasia are reluctant to communicate (see Historical Perspective). For instance, Cooper and Flowers (1987) noted that one of 25 Acquired Neurological Speech/Language Disorders in Childhood their subjects (10 years 3 months old) did not initiate any conversation and only rarely spoke during testing when assessed 6 years post-onset of anoxic encephalopathy.
On day 6 after the coma he was only able to write simple overlearned words, however, there was perseveration of letters. The legibility of his writing was good. His written naming was slightly better than his oral. In contrast to his written language skills his arithmetical abilities through the written mode was never impaired; however, he had problems writing numbers such as three hundred and four. As this was written 3–100–4, Van Hout and Lyon (1986) concluded that these errors were of a syntactical nature.
1981) found three right hemiplegic children to be markedly below three left hemiplegic children on the NWSST. A later study by Aram et al. (1985) comparing eight children with right hemisphere lesions and eight with left hemisphere lesions and two appropriate control groups showed that the children with right hemisphere lesions did not differ from their controls on any syntactic measure except for mean length of utterance. g. Mean Length of Utterance (MLU) and a Developmental Sentence Score (Lee, 1974)).